Mechanism: Botulinum toxins cause neuromuscular blockade by inhibiting the calcium-ion mediated release of acetylcholine at the motor nerve terminals, resulting in a diminished endplate potential and subsequent flaccid paralysis of the affected muscles. The paralysis persists until new nerve terminals form, usually within 2 to 4 months. Uses: given with haemaglutinin by local injection in the treatment of- hemifacial spasm, blepharospasm, spasmodic torticollis, lower limb spasticity in Children: with cerebral palsy, and upper limb spasticity associated with stroke in adults Botox: mainly used.
Vial
1610 EGP
Mechanism: Botulinum toxins cause neuromuscular blockade by inhibiting the calcium-ion mediated release of acetylcholine at the motor nerve terminals, resulting in a diminished endplate potential and subsequent flaccid paralysis of the affected muscles. The paralysis persists until new nerve terminals form, usually within 2 to 4 months. Uses: given with haemaglutinin by local injection in the treatment of- hemifacial spasm, blepharospasm, spasmodic torticollis, lower limb spasticity in Children: with cerebral palsy, and upper limb spasticity associated with stroke in adults Botox: mainly used.
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